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DR. James Jeremy Mccormick, PH.D.
Dr. James Jeremy McCormick is a researcher in human and environmental physiology whose research examines the physiological and cellular responses to environmental heat stress, with a particular focus on populations at increased risk of heat-related health effects. His research integrates whole-body human physiology with molecular and cellular approaches to characterize the mechanisms underlying individual differences in heat tolerance and susceptibility to heat-related injury.
Dr. McCormick’s research program focuses on defining the human heat stress response across multiple levels of biological organization, from thermoregulatory and cardiovascular responses to cellular stress and survival pathways. His work has examined the effects of heat exposure in older adults, including individuals living with chronic diseases, with the objective of identifying physiological and cellular mechanisms that may contribute to impaired heat tolerance in these populations.
A central component of his research investigates the cellular mechanisms involved in the response to thermal stress, including autophagy, the heat shock response, and apoptotic signalling. By integrating these cellular outcomes with measures of whole-body thermoregulation and cardiovascular function, his work seeks to elucidate how environmental heat stress affects cellular homeostasis and physiological function, and how these responses may be modified through heat mitigation and acclimation strategies.
Dr. McCormick completed his doctoral training in exercise physiology and molecular biology, where his research investigated cellular protective mechanisms associated with exercise in individuals with prediabetes. He subsequently joined the Human and Environmental Physiology Research Unit at the University of Ottawa as a postdoctoral fellow under the supervision of Dr. Glen Kenny, where he expanded his research program to investigate the integrative physiological and cellular responses to environmental heat stress.
His research contributes to a broader understanding of the physiological consequences of extreme heat and the biological mechanisms that may explain differential responses to environmental stress. This work has implications for developing evidence-based approaches to heat mitigation and acclimation and for informing strategies to reduce heat-related health risks in older adults and other populations with chronic health conditions.